US 11,839,195 B2
Parthenocarpic tomato plants with loss of function mutation in an AGL6 gene and methods of producing same
Rivka Barg, Rehovot (IL); Yehiam Salts, Rehovot (IL); Chen Klap, Kfar-Saba (IL); Itzhak Arazi, Kiryat-Ono (IL); Ester Yeshayahou, Kfar-Saba (IL); Anthony Bolger, Aachen (DE); and Sara Shabtai, Karmei Yosef (IL)
Assigned to The State of Israel, Ministry of Agriculture & Rural Development, Agricultural Research Organization (ARO) (Volcani Center), Rishon-LeZion (IL)
Appl. No. 16/071,097
Filed by The State of Isreal, Ministry of Agriculture & Rural Development, Agricultural Research Organization (ARO) (Volcani Center), Rishon-LeZion (IL)
PCT Filed Jan. 19, 2017, PCT No. PCT/IL2017/050078
§ 371(c)(1), (2) Date Jul. 19, 2018,
PCT Pub. No. WO2017/125931, PCT Pub. Date Jul. 27, 2017.
Claims priority of provisional application 62/281,227, filed on Jan. 21, 2016.
Prior Publication US 2021/0037779 A1, Feb. 11, 2021
Int. Cl. A01H 6/82 (2018.01); A01H 1/06 (2006.01); A01H 5/08 (2018.01)
CPC A01H 6/82 (2018.05) [A01H 1/06 (2013.01); A01H 5/08 (2013.01); A01H 6/825 (2018.05)] 11 Claims
 
1. A tomato plant exhibiting a facultative parthenocarpy having a loss-of-function mutation in a homozygous form in a coding sequence of SlAGAMOUS-LIKE 6 (SlAGL6) gene,
wherein said plant further exhibits (i) for fruits selected weighing minimum 8 grams each a fruit yield/plant at least about the same as that of a seed-bearing, non-parthenocarpic tomato of the same genetic background when grown together under identical conditions, wherein said about is defined as ±10%, and (ii) an average fruit weight/plant at least about the same as that of a seed-bearing, non-parthenocarpic tomato of the same genetic background when grown together under identical conditions, wherein said about is defined as ±10%, and wherein said loss of function mutation comprises (I) a mutation comprising a 175 nucleotide deletion, comprising the last 98 nucleotides of intron 1, all of exon 2 (76 nucleotides), and the first nucleotide of intron 2, or (II) a C268/t mutation in said SlAGL6 gene, said mutation resulting in a premature stop codon in said coding sequence.