| CPC C07K 14/7051 (2013.01) [A61K 40/11 (2025.01); A61K 40/31 (2025.01); A61K 40/4202 (2025.01); A61K 40/4204 (2025.01); A61K 40/421 (2025.01); A61K 40/4211 (2025.01); A61K 40/4215 (2025.01); A61P 35/00 (2018.01); C07K 14/70507 (2013.01); C07K 14/70596 (2013.01); C12N 5/0636 (2013.01); A61K 38/00 (2013.01); A61K 40/50 (2025.01); A61K 2239/23 (2023.05); A61K 2239/29 (2023.05); A61K 2239/31 (2023.05); A61K 2239/38 (2023.05); A61K 2239/48 (2023.05)] | 11 Claims | 
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               1. A method for treating cancer in a human subject while reducing host-versus-graft (HvG) rejection by an innate T cell or NK cell in the human subject, the method comprising: 
            administering an engineered cytotoxic T cell to the human subject, wherein the engineered cytotoxic T cell comprises one or more chimeric antigen receptors (CARs) comprising: 
                a first antigen binding moiety exhibiting specific binding to CD7, wherein the first antigen binding moiety comprises a first scFv comprising a heavy chain variable domain (VH1) comprising the polypeptide sequence of SEQ ID NO: 91 and a light chain variable domain (VL1) comprising the polypeptide sequence of SEQ ID NO: 90; and 
                  a second antigen binding moiety exhibiting specific binding to CD19, wherein the second antigen binding domain comprises a second scFv comprising a heavy chain variable domain (VH2) comprising the polypeptide sequence of SEQ ID NO: 88 and a light chain variable domain (VL2) comprising the polypeptide sequence of SEQ ID NO: 87, 
                wherein each CAR of the one or more CARs further comprises: 
              a hinge polypeptide, which is C-terminal to the first antigen binding moiety and the second antigen-binding moiety; 
                  a transmembrane polypeptide, which is C-terminal to the hinge polypeptide; and 
                  an intracellular signaling polypeptide comprising a signaling domain of CD3-zeta, which is C-terminal to the transmembrane polypeptide. 
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