| CPC C07K 16/2803 (2013.01) [A61K 45/06 (2013.01); A61P 35/00 (2018.01); C07K 16/2863 (2013.01); A61K 2039/505 (2013.01); C07K 2317/565 (2013.01); C07K 2317/76 (2013.01)] | 20 Claims |
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1. A method of inducing or enhancing an anti-tumor T-cell response, increasing T-cell proliferation, reducing cancer-induced suppressor myeloid activity, increasing natural killer cell cytotoxicity, increasing macrophage phagocytosis, increasing generation of M1 inflammatory macrophages, decreasing generation of M2 suppressor macrophages, increasing dendritic cell number in a tumor microenvironment, increasing dendritic cell activation, treating an HLA-G expressing cancer, or treating a MHC-I expressing cancer in a human subject in need thereof, comprising administering to the subject a monoclonal anti-immunoglobulin-like transcript 2 (ILT2) antibody or an antigen-binding fragment thereof, wherein the anti-ILT2 antibody comprises three heavy chain CDRs (HCDRs) and three light chain CDRs (LCDRs), wherein the HCDR1-3 and LCDR1-3 comprise:
a) SEQ ID NOs: 13-18, respectively, wherein X in SEQ ID NO: 15 is selected from A, C, and S;
b) SEQ ID NOs: 1-6, respectively; or
c) SEQ ID NOs: 7-12, respectively.
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12. A method for treating cancer by increasing efficacy of an anti-PD-L1- or anti-PD-1-based therapy against a cancer cell expressing HLA-G, MHC-I, or both in a subject in need thereof, comprising administering to the subject a monoclonal anti-immunoglobulin-like transcript 2 (ILT2) antibody or an antigen-binding fragment thereof, wherein the anti-ILT2 antibody comprises three heavy chain CDRs (HCDRs) and three light chain CDRs (LCDRs), wherein the HCDR1-3 and LCDR1-3 comprise:
a) SEQ ID NOs: 13-18, respectively, wherein X in SEQ ID NO: 15 is selected from A, C, and S;
b) SEQ ID NOs: 1-6, respectively; or
c) SEQ ID NOs: 7-12, respectively.
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