| CPC C07K 16/40 (2013.01) [A61K 39/4611 (2023.05); A61K 39/4621 (2023.05); A61K 39/4631 (2023.05); A61K 39/4636 (2023.05); A61K 39/46431 (2023.05); A61K 39/46434 (2023.05); A61K 39/464412 (2023.05); A61K 39/464495 (2023.05); C07K 14/7051 (2013.01); C07K 14/70514 (2013.01); C07K 14/70517 (2013.01); C12N 5/0636 (2013.01); C12N 5/0638 (2013.01); A61K 2239/22 (2023.05); A61K 2239/26 (2023.05); A61K 2239/31 (2023.05); A61K 2239/38 (2023.05); C07K 2317/622 (2013.01); C07K 2319/30 (2013.01)] | 18 Claims |
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1. A method of treating a tumor in a subject, comprising administering to the subject a T cell, wherein the T cell comprises:
a) a chimeric antigen receptor (CAR) comprising an extracellular antigen-binding domain comprising a single chain variable fragment (scFv) that binds to a first antigen that is expressed at the surface of a cell of the tumor, and an intracellular signaling domain that is capable of activating the immunoresponsive cell and comprises a signaling domain of CD28, and
b) an inhibitory chimeric antigen receptor (iCAR) comprising an extracellular antigen-binding domain comprising a single chain variable fragment (scFv) that binds to a second antigen that is not expressed on the tumor cell surface, and a signaling domain of an immunoinhibitory receptor selected from the group consisting of CTLA-4, PD-1, LAG-3, 2B4, and BTLA;
wherein binding of the CAR to the first antigen induces cytotoxicity of the T cell, and binding of the iCAR to the second antigen reduces the cytotoxicity of the T cell induced by the CAR.
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